The Shocking Truth About Eggs & Heart Disease

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Hello Health Champions today I want to talk  about the shocking truth regarding eggs and heart  

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disease there is a prevailing truth an accepted  truth in mainstream medicine and in our society as  

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a whole that cholesterol is bad that saturated fat  is bad that saturated fat increases cholesterol  

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and that's why saturated fat is bad and also that  high levels of cholesterol causes heart disease  

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and then basically we've been told this so many  times that we don't question it anymore that the  

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case is closed this is how it is and we're going  to talk about all of these different factors and  

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when I talk about it in a way that you learn  to think about it not just refer to some study  

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or some article but you'll understand these things  and when you think about them you'll be able to go  

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hey that makes sense or that does not make sense  but how come this became the prevailing truth the  

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mainstream truth the current Dogma and a lot of it  has to do with the fact that we associate things  

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that these fatty plaques have some cholesterol  in them and therefore we think hey that must  

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come from somewhere it must be from what we're  eating it must be the cholesterol and a lot of  

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that thinking had to do with something called  the seven countries study so this was late 50s  

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early 60s where a guy called Ansel Keys who was a  nutritionist was very convincing in his arguments  

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so here is how that study came about he studied  number of countries and as you soon notice there  

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were a lot more than seven countries and in fact  there were 22 countries that he started with and  

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then you would think this should be called the  22 countries study but he selected a number of  

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these countries so these were the only countries  that ended up in the study which is five of them  

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and then the other countries were scrapped because  they didn't fit what he was looking for so he was  

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had this idea that he was trying to prove that  diets high in saturated fat and cholesterol in  

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countries where they had those diets they also had  more heart disease so he had to pick the countries  

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that match what he was trying to prove and that  was only five countries out of 22 that matched  

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his criteria and then he had to go and find two  more countries Greece and Yugoslavia so now he  

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had seven countries where he could kind of see a  pattern and that's what he published as the seven  

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country study and you don't have to be a rocket  scientist to see that cherry picking like that  

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is not a great idea if you want to be objective  but even so this has been hailed as a landmark  

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study and is basically the foundation of why we  think the way we do today and since then there's  

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been hundreds of studies performed on these topics  and they tend to kind of fall 50 50 or in in two  

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opposite camps not necessarily equally split but  there are some studies that prove that show that  

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high cholesterol troll is strongly correlated with  cardiovascular disease but for every study that  

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says that there is another that says there there's  no correlation or even an inverse correlation that  

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actually high cholesterol High saturated fat diets  result in less cardiovascular disease and that's  

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why he had to scrap most of those 22 countries  because a lot of them actually showed the opposite  

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relationship but why is it so difficult to  figure this out if we have gold standard research  

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double-blind Placebo control why can't they figure  it out and one thing that we have to ask is who  

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pays for it and these studies are not cheap they  are in the millions or tens of millions of dollars  

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to perform a big comprehensive well designed  study so if you have tens of millions of dollars  

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sitting around you can go and order yourself  a study and someone will be happy to perform  

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it for you but for the most part the people or  the companies with that kind of money tend to be  

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pharmaceutical companies so they have huge budgets  because it's in their best interest to prove that  

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high cholesterol causes heart disease but here's  an astounding fact that most people don't realize  

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but it kind of makes sense if you understand human  nature we don't want to look bad so if someone  

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orders a study and they get the results back  and they're favorable then there's an 85 percent  

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chance that their studies will get published if  they can get it through the peer review process  

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to show that it's well designed and Rel relevant  then 85 percent of those will get published but  

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if it is not favorable if the results coming back  do not support what they wanted then there's an  

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85 percent chance that it won't get published and  these are not totally exact numbers because it's  

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going to vary a little bit but they're roughly  in the ballpark and what this shows us is that  

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what we call scientific truth that we base this  on double-blind placebo-controlled studies and  

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we think hey what's out there what's published  that represents the truth but what's built into  

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this process is a 45x bias toward the person who  ordered the entity who ordered the study so we  

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can't just look at what's out there and assume  that that's the whole truth and nothing but the  

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truth so I don't have tens of millions of dollars  sitting around either so I have performed some  

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of my own studies on a smaller scale and you may  have seen those videos where I do something I get  

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some blood work then I eat something and then I do  blood work again and then I report what happens so  

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for example I ate 100 eggs in seven days and the  pre and post blood work showed a few items going  

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up or down but overall I did not get sicker there  were no dramatic markers that I had a poor health  

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after doing that then another time I ate a hundred  tablespoons of butter in 10 days and interestingly  

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you would think that all that saturated fat  would end up in my bloodstream as triglycerides  

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but my triglycerides went down and my insulin  resistance improved after doing that I ate a  

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hundred hamburgers in 10 days or rather a hundred  hamburger patties because I didn't want to eat a  

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bunch of bread and carbohydrate to screw up my  metabolism and interestingly my cholesterol went  

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from 233 to 199 which is one of the lowest ones  I've had in recent decades and I don't really  

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know why I thought it would stay pretty much the  same and another interesting thing was the blood  

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urea nitrogen the bun which went from 16 to 10  even though I ate almost twice as much protein  

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as I usually do and the residue from protein is  this nitrogen that we can measure in blood urea  

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nitrogen and when I mentioned in that video that  I wasn't sure why that Bond went down and I still  

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don't one of my viewers enlightened me and said of  course it went down because you ate the hamburger  

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without the bond and if you notice I put quotation  marks around the word studies because I do that  

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kind of tongue-in-cheek I don't believe that these  are good examples of research this is actually  

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pretty bad science and I'm not claiming that it's  anything else and the reason is that the total  

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sample population meaning the number of people  studied is exactly one namely myself and it still  

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costs a few hundred bucks to do that so the reason  it's not great science is that I may not be like  

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you whatever results I get could be different than  yours but I'm not claiming that it's good science  

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or that I represent you but guess what when they  do bigger studies if they have a study with 10  

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000 people in them and then they have 55 percent  of people respond a certain way that's going to  

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create a trend it's going to be statistically  significant probably even though 4 500 people out  

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of the 10 000 didn't respond that way and I'm just  making coarse gross examples here to illustrate  

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something but the point is that it doesn't matter  how big the study is it may still not be like you  

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okay so you still have to figure out you and the  way that you do that is in these videos I explain  

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the mechanisms and the basic physiology so you can  start understanding how the Body Works rather than  

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just looking at one example whether it's mine or  somebody else's and then you can look at your own  

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blood work or you can look at your own results  in life how you feel your weight your waistline  

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Etc and then you figure you out but you can't  do that unless you understand some mechanisms  

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and some basic physiology let's look at some  very simple facts eggs have cholesterol eggs  

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have saturated fat but the question then is  first if we eat a bunch of eggs does this  

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mean that we raise our blood cholesterol we  don't know sometimes we do sometimes we don't  

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but if it did go up would that be a bad thing  and here's the huge misconception that a lot  

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of people think if it goes up then that's bad  but in itself it doesn't mean anything we have  

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to look at the bigger picture now let's  take a look at the concept of conversion  

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of turning one thing into another because based  on the prevailing truth about eggs cholesterol  

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saturated fat and heart disease as a culture  we believe that dietary cholesterol leads to  

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plaques leads to atherosclerosis so let's take an  example let's make a comparison so here we have  

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an egg which has obviously cholesterol and  then we have this theory that this leads to  

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plaquing and atherosclerosis and narrowing of  arteries and this is basically how we think  

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based on these studies the seven country  studies that the cholesterol from here ends  

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up as cholesterol in the plaque it's the same  cholesterol more of this leads to more of that  

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but here is a question now if we assume that  maybe this isn't true maybe there's some  

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conversion going on and let's ask a question  that if in another example that we have a cow

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and this cow has a body with some fat in it  with some saturated fat with cholesterol in  

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the fat this cow produces milk from which we  can make butter that has lots of fat lots of  

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saturated fat lots of cholesterol so if this cow  is full of cholesterol where did that come from  

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is it because that cow ate a bunch of eggs with  cholesterol did it eat a bunch of butter with  

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cholesterol did it eat a bunch of meat does it  go around eating the other cows is that how the  

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cholesterol ends up in the cow no if this cow eats  what it's supposed to then all it eats is grass  

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green grass pure carbohydrates basically it has  a trace amount of protein and and fat in it but  

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basically this cow eats things eats grass that has  no cholesterol and yet it ends up with all that  

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cholesterol in the body so maybe if we understand  that there's a conversion process maybe that  

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cholesterol in the plaque doesn't come from there  that's a possibility and this conversion is also  

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known as metabolism which means to transform so  it's a form of transformation so if we look at the  

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things that we eat as humans and we are by the way  quite similar to a cow in that we are both mammals  

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we both store fat and we store it as about half  saturated and about half mono unsaturated that's  

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typical for the body fat or both a cow and a human  so the big groups the big macronutrients that we  

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eat are protein carbohydrates and fat and protein  the purpose is to turn into tissue to building  

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blocks to body parts but the excess will actually  turn into carbohydrates and carbohydrates in turn  

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can become fat but it can't work the other way  around so we can convert things sometimes one  

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way but not necessarily both ways and then all  three of these turn into something called acetyl  

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COA and don't worry about the name don't try  to memorize any of this I'm just trying to show  

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you that we have something called conversion  metabolism transformation we eat these things  

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and along the way they become something else this  is an intermediary metabolite that we use to make  

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energy so if we make energy from protein carbs or  fat it's because we can turn it into acetyl COA  

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but if we're really really low on carbs this  acetyl COA can turn into something else depending  

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on circumstances depending on what's appropriate  for the body what it needs at that time so if  

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we're low on carbs we can turn this acetyl-coa  into ketones and this acetyl-coa is also the  

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raw material for cholesterol so again I'm just  trying to show you that this cholesterol didn't  

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necessarily come from cholesterol that we can turn  anything that we eat can turn into cholesterol if  

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we need it but here's the thing it's really not  a mystery anymore today we know what the cause of  

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cardiovascular disease is and it is chronic  inflammation oxidative stress and something  

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called insulin resistance I'm not going to go into  depth here because I've done videos on all of this  

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and it's also something called metabolic syndrome  or Syndrome X and this is a label this is a cons  

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steps when we're metabolically unhealthy then  we have certain manifestations and if we have  

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abdominal obesity hypertension meaning high blood  pressure if we have high fasting glucose if your  

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body can't control blood glucose as in type  2 diabetes if we have high triglyceride high  

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blood fats also very common in diabetes if we  have low HDL the supposedly good cholesterol  

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which of course there is no good or bad there's  just appropriate and appropriate cholesterol then  

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if you have any three out of these five then they  consider you to have metabolic syndrome and then  

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they know that you'll be predisposed to all these  different problems of heart disease Etc but the  

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people with metabolic syndrome what they really  have is chronic inflammation oxidative stress and  

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insulin resistance so these three things are the  causes that we need to work on and that we need  

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to control and this is very very well established  there's really no discussion or controversy about  

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this at all what there still is controversy about  is which type of foods and what type of Lifestyle  

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will create these states that cause cardiovascular  disease but the question now in this video is do  

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eggs cause cardiovascular disease so the question  is do eggs cause these three things and the answer  

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is no they don't there is nothing in there  that causes insulin resistance or oxidative  

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stress or inflammation unless you are allergic  to them then that becomes a bad food for you  

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or if you combine the eggs with things like sugar  margarine and chemicals and this is the problem  

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with a lot of observational studies like the seven  country studies that he picked countries that had  

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a high intake of saturated fat and cholesterol  but what else did they eat did they eat a bunch of  

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sugar margarine and chemicals how much did they  smoke do they ever exercise we don't know but  

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here's what we do know High total cholesterol in  itself does not cause heart disease and here's a  

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article that I've referenced once before but it's  so good I want to bring it back so basically they  

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looked at the total cholesterol levels they only  measured two things they measure total cholesterol  

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and how many people died and this was a huge study  because they did this in Korea over a decade or so  

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and it was basically everyone in Korea more or  less so 12.8 million adults were part of this  

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study and it's not like the seven country studies  where they don't know all the variables because  

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here they were only interested in two they didn't  care what else they did they only looked at what  

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happens with cholesterol and how many people  die of all the people who died what were their  

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cholesterol levels so they looked at 12.8 million  adults and they looked at all cause mortalities  

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what that means is they didn't really care if the  cholesterol caused heart disease or not all they  

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know because you could maybe save a few people  from heart disease but if they die faster from  

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something else then that's not really a gain is  it so all cause mortality is pretty much what  

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we're interested in and here's the interesting  thing that they found that we're told that high  

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cholesterol is bad low cholesterol is good and  they give people statin drugs I've had people  

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come through my office on statin drugs with the  cholesterol under 120 and they keep giving them  

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the stat drug as if less is always better but  here's what we find on this graph is that low  

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cholesterol is really really bad like really low  cholesterol is really bad and if we compare that  

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to very high cholesterol we see that a cholesterol  of of 110 120 is many many times worse much more  

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likely to cause death than a cholesterol of 300  but when was the last time you saw a commercial  

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for a medication to raise cholesterol it's like  oh my God your cholesterol is 120 we got to get  

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we got to get you up into the 250s they don't  do that so much do they because we have this  

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bad idea that less is always better and in fact  what we found in this study was that The Sweet  

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Spot the absolute lowest death rate the optimal  cholesterol would fall somewhere around 2 30. and  

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if we looked at the area where they had a very  slight increase above the lowest rate of death  

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like less than 10 percent increase above that  lowest rate we would consider that a pretty good  

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pretty good odds then that gives us a pretty wide  range where we are safe so anywhere from 180 to  

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270 is a very slight increase in risk and then  we overlap that with the standard range with the  

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normal recommendations that we get all over the  world is that the cutoff is a 200 anything over  

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200 is too high and you should take medication  to control it and if you notice here that  

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what the study found to be the optimal  range where you have just a slight increase  

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Falls almost entirely in the red area where you  would be prescribed or recommended medication  

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or lifestyle changes or both to lower your  cholesterol now let's just add one more factor  

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to this because I think it's a great study but  remember they only look at the total cholesterol  

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and the total amount of death all-cause mortality  but what we have to realize is that some of these  

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people with high cholesterol were healthy and some  of them were very unhealthy and the other things  

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that we just talked about that we know cause  heart disease that some of these people have those  

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factors and some didn't so what if we were to take  this population and I'm speaking hypothetically  

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now and we took all the people with abdominal  obesity and we took them out of here because  

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we're trying to find out how does cholesterol  affect death if we now correct correct for the  

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people who had abdominal obesity which we know is  a risk factor and we also take out the people who  

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have hypertension and high fasting glucose high  triglycerides and low HDL we know these are risk  

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factors for heart disease let's take these people  out so now we isolate more for what cholesterol in  

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itself actually does and we also correct we take  the people out who have high insulin levels now we  

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have a pure population where only cholesterol is  a causative factor of death and I believe not that  

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I've ever known that they've done this study but  I very strongly believe that if we corrected for  

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this then the curve would look like that it would  be almost flat and I don't know maybe around five  

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six seven eight hundred total cholesterol that it  might go up for some other reason but I think the  

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majority of people with cholesterol in the three  to four hundred range if we correct for this which  

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we can control through lifestyle then we will  not have an increased risk from cholesterol and  

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please hear me when I say that I'm not suggesting  that 350 is any better than 250 what I am saying  

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the number of total cholesterol by itself is not  what we want to look at now let's just go back to  

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the actual causes that we talked about and see why  do they cause heart disease or why is part of the  

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reason so when we have insulin resistance chronic  inflammation oxidative stress and poor metabolic  

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Health what happens in the body is that the LDL  changes it's not that LDL is bad but we can change  

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LDL into something bad if we have these factors  present because LDL is normally relatively large  

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I call it large and fluffy that's how your liver  produces it large and fluffy it actually comes  

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from even larger lipoproteins that that shrink  down and then if they're healthy they kind of  

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fall into this range but if we have these factors  these destructive factors present in the body then  

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they hurt they damage these LDL particles and they  shrink so now this inflammation causes damage the  

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inside of your blood vessels plus it makes these  small ldls unrecognizable you can't recycle them  

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properly and they're small enough to go to the  wrong places and create damage so what I have  

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found in clinical cases we have done hundreds of  these we do blood work on everyone we test the  

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size of these in everyone so we can follow up and  see what the changes are and when we put people  

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on a lifestyle change where they eat no sugar  and no seed oils no processed damaged vegetable  

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oils we give them a high quality diet meaning good  quality meat fish chicken good quality vegetables  

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low carb vegetables non-starchy vegetables and we  combine that with intermittent fasting meaning we  

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restrict their feeding windows so they don't  eat every hour of every day we feed the meat  

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vegetables and yes Eggs unless they are sensitive  to eggs then we take them out and we also give  

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them high quality fats we give them extra virgin  olive oil lots of butter lard Tallow coconut oil  

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Etc stable saturated fats plus of course extra  virgin olive oil which is mostly mono unsaturated  

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then what we find is we measure these sizes before  and after and we find that it goes the other  

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way that by making these lifestyle changes your  reduced inflammation and the oxidative stress and  

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the insulin resistance and now the body can start  to slowly cycle out clean out these small damaged  

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ldls and then the body as it makes new and healthy  ones then the proportion of these small ones get  

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fewer and fewer lesser and less proportion but  then of course the question is does that always  

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work does this work a hundred percent of the  time and the answer is no nothing works a hundred  

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percent of the time and I put only here kind of  as a joke because 90 to 95 percent is Sensational  

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and yet that is kind of where we're where we're  getting maybe not in the first couple of months  

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maybe not the first time around for a lot of  people probably two-thirds to 75 percent they  

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get dramatic changes like the first time through  but for others we might have to play around with  

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it and figure out what they're sensitive to  is there an autoimmunity thing is there other  

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things causing inflammation but we need to put  this in perspective the only 90 to 95 percent  

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because there's something called Placebo and in  a double blind can placebo-controlled study they  

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always have different groups and they give one  group a placebo meaning something that looks like  

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it's going to do something but there's nothing in  there there's no active component in there and yet  

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the placebo group always gets somewhere around 35  percent Improvement 35 percent of people notice  

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Improvement even though they didn't get anything  so 35 percent is kind of the Baseline that's what  

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you get no matter what because when people believe  that they're going to get better they get better  

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and a success medication only has to be better  than placebo so if it's a very large study all  

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they have to show is that it's statistically  significant that it's better than Placebo so  

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one or two or three or five percent may be better  than Placebo and that's a sensational success for  

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a medication and if a medication can show maybe  a 40 Improvement then they are super happy with  

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that that's a sensational result but we of course  are not happy with 40 so we try to always get as  

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close to a hundred as we can so now we want to  start looking at other sources of inflammation  

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and this is where it gets tricky because now it  gets a little more complicated and a little bit  

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more individual variation but there are things  like allergies there is other toxicity there  

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is autoimmunity and of course gut issues if  you have inflammation in the gut if you have  

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poor absorption if you have leaky gut now we have  lots of variables that might be hard to control at  

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least in the short term that starts affecting  these numbers but if we're patient enough and  

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we're willing to continue to make changes and  monitor blood work and and check on these then  

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very often we can get to 98 99 and that doesn't  mean necessarily complete remission or that there  

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are no symptoms left but that we are much better  off than we were and then there is that last one  

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to two percent and this is very unfortunate but  some people are just given some bad cards that  

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there are various genetic factors that if you do  all the things we talked about you're still going  

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to be way better off but that may still not be  enough so the shocking truth about eggs and heart  

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disease is that you have been lied to we have  all been lied to for much too long for decades  

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they have been focusing all their attention on the  wrong guy they've been demonizing the hero because  

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it's not about total cholesterol it's not about  dietary cholesterol it's not about saturated fats  

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or how many eggs you eat it is about metabolic  health and all the factors that go into that like  

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insulin toxicity gut health and the list goes on  and on and that's why we need to understand more  

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about how the body works and how the world has  changed because the world has changed more in the  

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last 50 years than it has in the previous fifty  thousand and we need to understand how does that  

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affect us so that we don't just walk like lamps  to the slaughter so the solution is learn what  

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real food is and eat that and this includes eggs  as many as you like make sure to get good quality  

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eggs get the pastured eggs and eat both the yolk  and the egg white unless you have been found to be  

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sensitive or allergic to eggs because then they're  going to create inflammation and then they're bad  

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for you so everyone is different learn what works  for you but understand it from the mechanisms and  

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