Intermittent Fasting: Destroying Your Heart?
Hello, Health Champions. Can fasting actually increase the risk of heart attacks? Well,
according to a new study presented at a conference by the American Heart Association,
there is a 20,000-people study where they found that if you eat your meals in a
period of less than eight hours per day, as opposed to the normal 12 to 16 hours,
then there would be an increased risk of heart attack of 91%. This would come as quite a shock
to all the millions of people who are practicing intermittent fasting for the opposite reason,
to actually save their heart and improve their metabolic health. So, let's talk about this.
Well, there are some serious limitations to this. First of all, it is not a study yet; it's
abstract, like a preliminary study. It has not gone through the peer review process, meaning that
experts actually look at this and validate the data, and that the conclusions and the reasoning
are sound. Secondly, the data is self-reported, and they sent out a survey and asked people a
couple of times during a several-year period, and they ask, "Do you remember what you ate?" Even the
author of the study admits that this depends on recall and therefore may be inaccurate.
But perhaps the biggest problem with this is that they don't survey any other factors; they
don't control; they don't ask about anything else other than the time period during which food was
consumed. So, there are some factors of critical importance, such as what kind of food do you eat,
what sort of nutritional value does it have, but they also excluded factors such as sleep,
stress, exercise, and smoking, which have huge impacts on heart health.
But here's where we really need to question the validity of the study and their conclusions
because the study, the data was collected during the period from 2003 to 2018, and then in the
study, they reported it as intermittent fasting. But intermittent fasting is a very new concept,
even though certain cultures have practiced it for thousands of years. The concept of
regular intermittent fasting wasn't really a thing until the late 2010s,
which means that it was either after or toward the very end of when this data was collected. So,
there was probably no one in this study, or extremely few people in the study surveyed,
that thought they were actually practicing intermittent fasting on purpose.
And then you have to ask yourself, if they're not consciously practicing intermittent fasting,
then why are they doing this? What type of person would be eating during a shorter period of time
if they're not consciously doing intermittent fasting? And my guess is that this would be
something like a type A personality, someone who is very driven, very busy. They're probably
too busy to eat, and that's the reason that they skip breakfast and eat during a very short period
of time. They probably skip breakfast; they may even be the type of person that just goes
through the first half of the day on cigarettes and coffee, maybe the occasional donut. And when
they're very short for time, chances are that they're going to frequent fast-food places.
So, I don't know this for sure. This would be my suspicion. But the fact is, the people in the
study don't know this either because they didn't ask. Now, what's really disturbing to me is the
fact that this study isn't even published, and yet the American Heart Association jumps on it, and
then as a result of the authority of the American Heart Association, now of course, all the media
like NBC News are going to cover it, Washington Post, US News and World Report, USA Today,
and New York Times. I think there were about 6,000 different instances that had restated this study,
and then, of course, all the people writing for these news media, they're not health experts.
They are not versed in critically evaluating or assessing such a report. No, they just want to
create some sensationalism and tell everybody about the dangers of intermittent fasting.
So, now millions and millions of people who had their hope in getting healthy from intermittent
fasting now get really scared. What are you supposed to believe? And what's also disturbing
to me is that to me, it seems like the American Heart Association is not anchored in science
because there is no foundation in physiology. There's no way to open up a physiology textbook
and read something that makes sense to why this study would have that particular outcome. And if
it doesn't match physiology, then it should be the very last study that they bring out prematurely,
not the very first one. But yet, they jump at the chance to get this information out
there. It's as if they're just waiting for an opportunity to silence the voices of people who
do things differently than mainstream medicine. And whenever there's a movement that threatens the
American Heart Association and the establishment because people can get healthy on their own,
then of course, that's not good news for the American Heart Association or anyone
in that establishment because most of these organizations and most teaching hospitals and
research universities depend on funding, and a lot of that will come from Pharma,
who depends on a system where they can distribute their medication.
So, we always have to ask ourselves why they jump so quickly on this information,
and we have to ask ourselves who is involved, who has anything to gain or
lose by this information. And unfortunately, what's so disturbing is that a lot of times,
it comes down to politics, power, and finances rather than patient health.
Another reason they shouldn't jump on the premature study is that there is now abundant
evidence in just a few years about the power of intermittent fasting, on how, if you eat real
food and you time-restrict your eating, then it will very reliably and very consistently
improve all markers of metabolic disease that are related to cardiovascular disease. And when I say
all markers, I don't mean every single marker in every single person every single time. I mean that
on a study scale, we consistently improve these markers for the majority of people.
But that is also why you don't ever want to trust anybody's opinion explicitly, not mine,
not the American Heart Association, because everyone can be different. So therefore,
what you need to do is you need to study health, not just read lists off of the
internet. You want to watch some videos that will actually teach you mechanisms
to help you understand the body and some of the basic mechanisms in the body until you
can see the information on the internet come together. When does it make sense? When does
the information confirm other information? When do you see the trend and the tendency?
And then you want to do your own study with an N of one, and that means that the sample
size is one person, you. Normally, in terms of research, that's a joke because that means if
you only have one person, then it's not statistically significant. But when you
do your own study on yourself, it's the most significant because you are learning about you.
And then you want to measure your actual risk markers, the real markers for heart disease,
the risk factors. And I'm going to share those with you in just a second. Then, of course,
you do something, you change something, so you limit your feeding to 6 to 8 hours a day. And
it's probably not absolutely necessary that you change what you eat so much,
even though I would strongly recommend it because your chances, the probability
of getting good results, are going to go up dramatically if you start eating real food,
which means you cut back on sugar and soda and ice cream and cookies and all the packaged foods
that are basically primarily made up of sugar, flour, and seed oil.
And if you do that, if you start eating real food,
you will automatically cut out at least half of your carbohydrates,
maybe more. And you don't have to go into ketosis or go extremely low carb for any of this to work.
The fasting works in itself, but your chances are going to go up if you eat real food.
And then you keep that up for three months, and then you run those blood tests again to
reassess all those markers. And what is it that you're going to measure? What are the
real risk markers for heart disease? Well, they fall into two categories:
things that measure insulin resistance and things that measure inflammation and oxidative stress.
So, the first few markers are glucose, A1C, and insulin. Glucose is how much blood sugar you
have at any given moment. A1C is the average glucose over a 3- to 4-month period of time,
and insulin is the hormone that controls glucose. So when your glucose goes up,
your insulin goes up correspondingly to bring the glucose down. But when you become insulin
resistant, then it takes more and more and more insulin to keep the glucose down.
So now, there's something called HOMA-IR. And what you do now is you multiply the glucose by
the insulin and you divide by 405 if you measure glucose in milligrams per deciliter. If you
measure in millimoles, now you divide by 22.2. And the result is a number called HOMA-IR, homeostatic
model assessment of insulin resistance. And this would be the strongest indicator there is of
your risk of heart disease because, by far, the strongest correlation to heart disease is type 2
diabetes, and this is the best marker to assess your risk or your tendency of type 2 diabetes.
Some other strong indicators of insulin resistance would be your triglycerides,
how much fat is floating in your blood because if you're insulin resistant, it stays in the blood
for a long time. If you're insulin-sensitive, your cells will absorb it and use it. Waist
size is another strong indicator because once you get insulin resistance, you get fatty infiltration
of the liver and your abdominal cavity, and your belly grows. Another strong indicator is
hypertension or high blood pressure, which is also very strongly associated with insulin resistance.
Measuring inflammation and oxidative stress is not quite as straightforward,
but we have some really good indicators. HDL and LDL cholesterol will give us a good indication,
and it's not about how many milligrams you have of each, but it's the ratio because when the ratio
goes down, meaning there's less HDL in relation to LDL, that tends to indicate inflammation. We also
want to look at the size of the LDL particles, and you have to run a test called an NMR test to get
this information, but it's well worth it because you're assessing the actual risk factors. What
you're looking for on the test is to have what we call large and fluffy LDL particles because
a lot of inflammation and oxidative stress will shrink and increase the density. So,
if you have small, dense LDL particles, then that is a strong risk factor for heart disease.
One more factor we can measure is called high-sensitivity C-reactive protein,
which is a general inflammatory marker. Now, these are not the only markers,
but they are the main ones. And if you really want to dig into this and start
understanding your blood work in terms of how the body works, I created a blood work course,
and I'll put a link down below for that, and we can also get you some discounted
blood work where we measure all of these factors plus a whole lot more.
But the biggest problem in making change is that there is so much attachment to the status quo.
There are so many people, so much money invested in keeping things the same. So, when something
like low-carb or intermittent fasting becomes popular because hundreds of thousands, millions
of people see improvement, they get healthy, they make changes that they've never been able to do
before, that threatens this status quo. So now, people desperately cling to their old beliefs and
they say, "Well, we admit that you can reverse diabetes, we admit that you can lose weight,
we admit that it's really good for reducing visceral fat, but we don't know the long-term
effects." That's kind of their fallback that will save the day for them in any situation.
But that is why it's so important we understand the real risk factors because if you measure those
and they get better, then you are getting healthier. And this is both short-term and
long-term because so often what they're saying when they say, "We don't know the long-term
effects," what they're saying is, "Well, it might be good for your short-term, but it's probably
dangerous long-term." And that's not how it works. If we address and improve the real risk factors,
and the exception is if we use some chemical, some medication to suppress body physiology. If we just
bring in things that the body wants naturally, and we let the body do its thing, and the body
uses its infinite intelligence to achieve homeostasis, to regulate all these thousands
of variables to find that best balance, now this holds true. When you improve the markers, you are
getting healthier. But if we're not allowing homeostasis to do its thing, if we introduce
chemicals that interfere, that suppress certain markers, that block what the body is trying to do,
now we're not allowing this balance, and now we're creating a stress load and a toxicity on the body.
And this is probably why a lot of people have such a hard time understanding this because
we're trained in the idea that medication will make us healthy, and medication can't do that.
Medication is not always a bad thing, but it cannot restore the thing that isn't working.
All disease is because of something that isn't working right, and if we can address the root
cause, if there's enough healthy tissue and function in the body that we can support it
and bring back that function, now we get healthy. If we don't have enough balance,
enough tissue, there is sometimes a good idea to use medication, but it's only going to control
something at the expense of something else. It will not restore the original function.
So here is, in my opinion, the real risk factors and how important they are:
We have insulin resistance, then we have gut health and other sources of inflammation like
leaky gut and autoimmunity and food allergies and so forth. Then we have LDL particle health:
are they large and fluffy, or are they small and dense and damaged by inflammation and oxidative
stress? And then we have total cholesterol, as in measured by milligrams per deciliter or millimoles
per liter. And here is my belief, this is my opinion. I don't have any data to support these
specific numbers, and I just kind of grabbed them to give you an idea of how I feel about this. So,
I think insulin resistance is by far the strongest correlation, the strongest risk factor for heart
disease. It probably accounts for about 80% of that weighted importance. Then I think that
gut health and other sources of inflammation probably account for another 10%. I think the
LDL particle health, particle size, is another 9% or so. But keep in mind that most of the LDL
issues have to do with insulin resistance and other sources of inflammation, so this
is not really an independent risk factor; it's dependent on these two other factors. And then,
maybe total cholesterol accounts for maybe 1%, but this is only in my opinion for people
with very very large numbers. I don't think a number of 300 in itself is a risk factor if
everything else looks good. I think maybe, and I'm saying that because I don't know,
if your cholesterol gets up to 600-800, then you have some severe metabolic imbalance, some
genetic imbalance that can predispose you, but for most people, that is not going to be a factor.
This stuff can get really confusing when you learn something and you believe it,
and then they publish a study or they talk about a study like this that contradicts everything that
you believe. So, your proof is going to come from your results and your understanding. So,
if you go through and you do your own trial study on yourself, a three-month trial,
pre- and post-blood work, then the question is, what happened? That's going to be your proof.
So, if there are no significant changes in most of these variables,
then chances are that your issue is a little bit more complex. It's going to be something
like autoimmunity or inflammation or maybe you have a chronic infection or
some emotional trauma. The variables are unfortunately pretty endless.
It could also be that you get your test results back, you've gone through these changes,
and all or most of your markers are worse. If that's the case, then intermittent fasting is
probably dangerous for you; it's something you should stay away from. But if that happens,
please contact me and report to me on that because I'd love to know what planet you're from.
Well, kidding aside, we never want to say never, so it is possible that it could happen because
there are billions of people on the planet. There's probably a handful that would have some
extreme, inexplicable reaction, but it's going to be so rare that it is extremely unlikely to
happen. What's much more likely, and I would say is probably the case for more than 90% of people,
is that you would see all or most of your markers improve if you haven't
done intermittent fasting or low carb or some healthy living already for a period
of time. If you're just kind of new to this and you go through this process,
then I would say that all or most of your markers are probably going to show some positive change.
And then, if that's the case, then intermittent fasting, like for most people, if you can use
it to reduce insulin resistance, to improve metabolic health, that is the best thing that
you could do to improve your health, to reduce the risk of heart disease, and save your life.
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