I Ate 100 TBSP Of BUTTER In 10 Days: Here Is What Happened To My BLOOD
This is me loading up on butter at the grocery store because in the next 10 days I'm gonna eat
100 tablespoons of butter to see what all the saturated fat will do to my blood. Hello Health
Champions. Today we're going to talk about fat In the blood also known as triglycerides
and about the blood work that I did before and after eating 100 tablespoons of butter in 10
days and I did this because I wanted to see what would happen we keep hearing about how
bad fat is how saturated fat will clog up your arteries and increase your blood fats and cause
insulin resistance and diabetes so I wanted to do something sort of extreme just to see what would
happen I went and looked at some of the websites about how to get answers what causes high levels
of fat in the blood and the answer was pretty straightforward most people have high blood fats
because they eat foods with too much fat and they say the reverse is true also how do you treat this
how do you undo high levels of blood fats and that is of course the best way the number one
way is to eat less fat and here's what I ate during the 10 days 1420 grams of butter but
of course you have to eat some other things as well so I ate some meat and some fish and eggs
and lots of vegetables with that but all in all I eat 2 200 grams of fat or 220 grams of fat per
day and most of that was saturated 1170 grams of saturated fat so that saturated fat accounted for
42 percent of my total calories now if we check with some general guidelines they typically come
in around 10 percent of saturated fat and one in particular the American Heart Association says
that we should eat no more than five percent or maybe six percent of our calories from saturated
fat so basically I ate a little more than eight times as much saturated fat as you're supposed
to and I was thinking that really has to create some damage right and I agree with them that high
blood fats are not a good thing high blood fats can cause problems heart disease and pancreatitis
diabetes strokes and obesity are associated with metabolic problems where we see high blood fats
the question is is this caused by eating too much fat now in a little trial like this there's three
possible outcomes either my blood fats would go up as the standard model would predict and if
that's true then it seems like the standard model is pretty much correct that we should list listen
to that and we should follow that however if my blood fats would go down or stay the same then we
worry that there's something seriously wrong with that standard model or at the very least that it
is incomplete and doesn't really account for all the variables that matter now please hear me when
I say that more isn't better and just because I had certain results doesn't mean that you would
have those same results you need to understand the mechanisms and the things that I will talk
about and apply them in your case so that you can have an outcome that you want eating a hundred
tablespoons of butter is not something that you want to do long term because it is too limiting
it's called a stunt and I did it to hopefully get some people's attention so that we can start
thinking and questioning some of the guidelines which ones are true and which ones do we need
to modify and when it comes to fat and lipids in the blood we need to understand how does it work
how does the body transport them how does the body regulate them what are the different factors that
make blood fats go up or down what's the basic purpose first of all fat and water doesn't mix
so if you put a drop of oil in water it's going to float on the surface pretty much forever
and the same thing holds true in the body the blood is mostly water so we can't just put the
fat straight in there because it would Clump together and it wouldn't go anywhere so in the
body there's something called lipoproteins where we have proteins that carry the lipids and they
look pretty much like this this is a lipoprotein and they've solved this issue of water solubility
beautifully because there is a membrane on the outside and these green things those are
the heads of phospholipids so the green portion is water soluble and those yellow little things
are fatty acids sticking to the inside so the outside of this is water soluble and the inside is
fat soluble now these are still super super tiny they're measured in nanometers and on the inside
they carry the triglycerides and the cholesterol and we have different kinds so we have VLDLs, LDLs
and HDLs basically and we want to think of these in terms of size that the VLDL is extra large in
nanometers it measures about 40 to 80 nanometers the LDL is like a medium size about 19 to 23
nanometers and the HDL is small or extra small 5 to 15 nanometers so when something is extra large
it's because it contains a lot of fat this piece in the middle here with the fat the triglycerides
and the cholesterol that part is huge and then as they get smaller they contain less fat in the
middle in proportion to the surface membrane and the proteins which are these colorful balls and
the body makes these lipoproteins to distribute and regulate fat and cholesterol in the body so
we start off with a really big VLDL, very low density lipoprotein and this one is jam-packed
with triglycerides with fat and along the way its purpose is to distribute this and it's so full of
triglycerides that it's basically this leaking these fats all over the place and that's a good
thing because it's delivering fuel to the cells of the body and it delivers mostly triglycerides
by far but it also has a little bit of cholesterol to deliver and 90 percent of all the triglycerides
that you find in the bloodstream are going to be carried by VLDL so when we measure triglyceride
on a blood test it is really indirectly these lipoproteins we're measuring and primarily VLDL
so the VLDL and the triglycerides are going to be almost the same indicator but with a
different units and then what happens as this is floating around in the bloodstream then it's
going to release a lot of this fat along the way and the cells will soak it up and then this cell
this lipoprotein shrinks so it goes from being a VLDL to a regular LDL and at this point it's
going to be about 21 to 23 nanometers it's still going to be a good size it's medium but it's on
the large size and this is a large fluffy LDL this is what they're supposed to be but if this
LDL sticks around and it takes a few more laps and there's a lot of stress there's a lot of oxidative
stress and inflammation and too much sugar in the bloodstream now this LDL gets damaged along
the way and it shrinks a little bit further and now it becomes an oxidized LDL or a damaged LDL
and now we're going to see it at about 19 to 20. so the more of these small ones you have the more
damage there they can cause but the whole point is the VLDLs become LDLs after they unload their
cargo the other lipoprotein that's manufactured in the body it's called an HDL and it starts out
super super tiny and then as it goes around its job is the opposite of the VLDL. The VLDL unloads
and the HDL collects so the HDL is supposed to increase in size as it collects and picks up
some triglycerides but mostly cholesterol that has been used in the body and it brings it back to the
liver for processing and recycling so we want the VLDLs to be small indicating that they have
unloaded their cargo we want the LDLs to be large indicating that they have not been damaged and we
want the HDL to be as large as possible indicating that it's actually doing its job of picking up
that extra cholesterol there's a lot of confusion about how this is associated with heart disease
because most people and most doctors believe that high triglycerides and high LDL always go together
and this is true in some cases it is pretty common but it is not the way it needs to work
this is what it looks like when it's associated with high insulin when we have metabolically poor
health then yes we will see high LDL High total cholesterol high triglycerides at the same time
but only in the presence of high insulin what we see a lot when we start changing things around
when we're metabolically healthier it is that we can have high LDL and low triglycerides and
still be metabolically healthy and the reason is that we now have low insulin so in the presence
of low insulin we see the opposite relationship between LDL and triglycerides so triglycerides
are a very good indicator of insulin resistance of metabolically poor health now there's a lot of
confusion about why triglycerides and VLDL build up and get elevated and the number one reason
reason and really the only reason that they build up is that they are not being used the body makes
them for a reason they're there to provide fuel but they're not being used up because
there's too much of something else that has to be used first and here's how this works
first we eat some food and the fuel that we derive from the food can be in two forms it can either be
fat or it can be a carbohydrate and if we eat the fat now it can go directly into the bloodstream
and it floats around as fat or triglycerides like we talked about and it can be available as fuel
for the cells needs no conversion or we can eat carbohydrates and this carbohydrate gets into
the blood faster that's what carbohydrates do and they raise blood glucose and if I had a penny for
every time I heard that carbohydrate or glucose is the preferred fuel I'll be very wealthy but
it's not that it's the preferred fuel it's the fuel that has to be used up first because high
blood glucose is dangerous to the brain and it is very very tightly regulated because it causes
damage when it gets too high that's why if the body has the choice between fat and carbohydrates
it's always going to use the carbs first because they can cause damage if they go to high levels so
the more carbs you eat the less likely that you're going to use the fat that is also floating around
in the bloodstream at the same time but it doesn't stop there you can use some of the glucose some of
the carbohydrates you can use in the moments you can store a little bit but most of it it has to
be turned into fat so you eat the carbohydrate becomes glucose and then that turns into fat so
now we have two sources of fat we have the fat we ate and the carbs we ate they both float around
as fat except we have to use the carbohydrates first so there's no reason we can't ever get to
the fat but there's one more factor that makes this situation even worse and that is once the
carbohydrate is inside the bloodstream it needs some help to get that glucose into the cell and
there is a hormone called insulin and that opens the gate between the bloodstream and the cell to
let that glucose in so short term that's fine but if we eat a lot of carbohydrates then this insulin
is going to be chronically elevated because it's all ways trying to get that glucose into the cell
so now the insulin is pushing it into the cell but it also is preventing that fat from being used up
so now we have on so many levels it's difficult for the body to use that fat and that's why it
builds up however if we don't eat the carbohydrate if we eat primarily fat then the body is going to
start burning the fat first and whatever fat we put in the bloodstream gets used up and our VLDL
and our triglycerides never rise and there are endless lists of food that they suggest that we
can eat that they are good to lower triglycerides and lower blood fats and I'm not even going to go
through all these there's dozens on this list and there's hundreds on various different lists
but what I want to point out is that these Foods seem very very random we don't get any explanation
as to why these Foods would do that what do they have in common so I want to ask why what
is it that these foods are supposed to do and here's the answer if they don't raise glucose
and if they don't raise insulin then they're okay they could be beneficial for other reasons if you
don't have a glucose or a triglyceride problem but if you put it on the list to lower triglycerides
that's the only thing that you're concerned with how is it going to affect glucose and Insulin so
here's where I ate during these 10 days I kept a spreadsheet and I had 78 of my calories from fat
15 from protein and seven percent from carbs so the standard model the general wisdom of the day
would say it would predict that my triglycerides would go way up because I ate so much fat almost
80 percent and that would also be reflected in the vldl we're going to look at both markers and then
it would predict that my insulin resistance would increase because that is what they
say and also that because fat is such a strong contributor to obesity then I should also gain
some weight and here's what happened to my insulin resistance markers my weight went from 185 to 185
no change at all and I might add that I did not have time to exercise a whole lot I did a few
push-ups I went for a couple of walks but that's about it my glucose started off at 95 and after
10 days it was down to 84. my insulin started at 3.5 and it went down to 2.8 and I might add
that an ideal range for insulin is between two and five whereas a diabetic is going to be more on the
range of 25 to 30. and if you calculate something called HOMA-IR or homeostatic model assessment of
insulin resistance you're taking into account the glucose levels but also how much effort how
much insulin it takes to keep the glucose levels down so you multiply the glucose by the insulin
you divide by 405 and an ideal number would be somewhere around one so anything from 0.5 to 1.5
would be ideal and very insulin sensitive and I started out at 0.82 and after 10 days of eating
butter I was down to 0.58 even though we're supposed to increase insulin resistance with
saturated fat and I ate eight times as much as I was supposed to again nothing I recommend but
obviously does not have that predicted effect of worsening insulin resistance and then let's look
at normal and healthy levels of triglycerides so these numbers come from Kaiser Permanente that's
an insurance company that ensures people they look at various different risks and they know that if
people have high triglycerides then they're very risky to ensure they say that optimal for an adult
is less than a hundred milligrams per deciliter and then they also say that younger people like
teens would be less than 90 and pre-teens would be less than 75 so the general idea is that these
numbers naturally and normally go up as we age but I would rather say that younger people have
less because they haven't lived long enough to get clogged up and congested whereas if you keep
burning through the fat that you eat by keeping insulin down there's no reason we shouldn't have
these low numbers all the way through our lives and they also say that a normal risk not optimal
or ideal but just kind of average normal would be 150 to 199 but keep in mind most people are
metabolically challenged challenge these days they have some degree of insulin resistance
so let's take a look at my numbers and again optimal is less than a hundred so I started
off at 56 and after eating butter after stuffing my bloodstream with saturated fat for 10 days it
was at 54. and again these triglycerides are carried by the vldl so we confirm this finding
the lab range would be 5 to 40 with anything over 20 being way too high in my opinion and I started
off at nine and after 10 days with butter I was still at a nine so I think we need to challenge
the standard Model A little bit and understand that it's not about the fat it's not about the
saturated fat it is how high we're driving the insulin with the other Foods we're eating at the
same time I think Mark Twain says it best what gets us in trouble is not what we Don don't know
it's what we know for sure that just ain't so and we've heard these things so long that they become
truths even though it may not be so so keep in mind we want to focus on the key factors we
want to look at insulin and VLDL when it comes to understanding fat in the blood and we don't want
to focus on LDL by itself we want to understand healthy versus unhealthy LDL but the number itself
means very very little and it is not about how much saturated fats you eat eat quality food of
a wide variety and understand the mechanisms as they apply to you but now here's the thing one
of the best ways to find that perfect balance for you is to know your Baseline and if you didn't see
last week's video that's where I was talking about how you can do blood work and if you understand
what those markers mean you can catch imbalances much much sooner and you can get a truer picture
of where you are for example if you're more or less insulin resistant you can also find out
some markers that most people don't test for most doctors never test for things like homocysteine or
ferritin and if homocysteine and ferritin are high then you can actually aggravate insulin resistance
you can promote insulin resistance simply by those two markers being elevated and because most people
have no idea and unfortunately most doctors have no idea I'm about to launch a a blood work course
where I'm going to teach over eight weeks and help you understand your blood work so that you
can truly take charge of your own health get some blood work and understand what it means so you can
get a baseline make some changes and then monitor the results and understand it better than you ever
have before so if you check out the links down below they'll take you to where you can find out
and learn more about that course if you enjoyed this video you're going to love that one and if
you truly want to master Health by understanding how the body really works make sure you subscribe
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